[潤稿] 4500_英潤英_生醫類_12096字_20200715
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[必]工 作 量： 12096 字（word）
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[必]截 稿 日： 20200715
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a.
It was previously shown, that exosomes secreted by cancer cells will
sufficiently promote metastasis by various ways. For example exosomes derived
from breast cancer and prostate cancer cells induce neoplasia through
transfer of their miRNA cargo [14,15]. The plasticity of cancer cells may
also be attributed in part to exosomes, with exosomal miR-200 from metastatic
breast cancer cells enhancing the epithelial to mesenchymal transition (EMT)
and metastasis of otherwise weakly metastatic breast cancer cells [16]. EVs
participate in intercellular mitochondrial transfer. Horizontal transfer of
mitochondria occurs through the transfer of either mitochondria-derived
vesicles or intact mitochondria. To date, the mechanisms of intercellular
transfer of free mtDNA across both mitochondrial inner and outer membranes
and the plasma membrane remain elusive.
b.
Mitochondrial content, especially damaged or oxidized components resulting
from IR exposure, can act as damage-associated molecular patterns (DAMPs)
that activate an inflammatory response when present in the cytosol or
released from cells [43]. The mitochondrial antiviral signaling protein
(MAVS), located in the mitochondrial outer membrane, an innate immune
signaling molecule, is involved in radiation response via its oligomerization
mediated by radiation-induced ROS. MAVS is involved in IFN-β and
IFN-stimulated gene expression in the response to IR. MAVS complexes
promoting the nuclear translocation of the NF-κB and IRF3/7 transcription
factors, respectively, and thus elicit the innate antiviral response.
[選]其他事項：無
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※ 文章網址: https://www.ptt.cc/bbs/translator/M.1594280445.A.843.html
※ 編輯: lockq (140.120.25.103 臺灣), 07/09/2020 15:41:33
※ 編輯: lockq (140.120.25.103 臺灣), 07/09/2020 15:42:19
推
07/09 19:13,
3年前
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推
07/10 10:24,
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※ 編輯: lockq (125.230.253.207 臺灣), 07/11/2020 08:57:48
※ 編輯: lockq (140.120.25.103 臺灣), 07/15/2020 15:15:42
※ 編輯: lockq (140.120.25.103 臺灣), 07/15/2020 15:16:09